Reynaud’s phenomenon [1] [2] is a problem of blood supply to the extremities. The origins of RP have not been clearly identified. In some cases, it is due to emotional stress and it is often associated with smoking cigarettes; however, in common with many common morbidities, it must have neural, autonomic (ANS), and/or multi-sys- temic origins. It is considered to be a problem of the ANS, in particular the sympathetic nervous system, which causes vasoconstriction of the peripheral blood vessels which often leads to hypoxia in which regions of the body are deprived of an adequate supply of oxygen. It affects the connective tissues, the thickness and colour of the skin which may turn white or in extreme cases blue, and the temperature of the extremities [3] [4] .

The pathological indications which accompany Raynaud’s phenomenon include connective tissue disorders e.g. scleroderma, ulcerative processes, raised or lowered (idiopathic) blood pressure and/or cardiovascular dis- ease(s) e.g. atherosclerosis, problems of the peripheral nervous system and hence loss of feelings in the extremi- ties, rheumatoid arthritis, etc.

There is not currently a single rapid test which can be used to diagnose the onset and/or progression [5] of Raynaud’s phenomena because the condition is a complex multi-systemic condition.

Secondary Raynaud’s phenomenon often leads to sores or gangrene (tissue death) in the fingers and toes which can be painful and difficult to treat. In severe cases, there may be a requirement to treat skin ulcers and serious tissue damage, and/or for surgery including amputation.

The current mechanism for diagnosis of Raynaud’s phenomenon involves a complex set of diagnostic tests and criteria. This is summarised by [6] Maverakis, Patel and Kronenberg. Individual tests which are involved in making a diagnosis include:

・ Digital artery pressure: by measuring the blood pressure in the arteries of the fingers before and after the hands have been cooled. A decrease of ≥15 mmHg is considered to be confirmation of the condition.

・ Ultrasound: to assess blood flow.

・ Blood count: to assess whether anaemia or kidney disease may be a problem.

・ Blood test of urea and/or electrolytes to assess whether kidney dysfunction is an issue.

・ Thyroid tests.

・ Antibody screen looking for signs of rheumatoid arthritis or inflammatory response (via erythrocyte sedimentation rate and/or C-reactive protein).

SVS is based upon the observation that changes of colour perception have pathological origins and hence that measurements of colour perception could be related to a spectrum of pathological indications. SVS has been used to diagnose a wide spectrum of almost every conceivable disorder or combination of pathologies including, but not limited to, diabetes, cardiovascular disease, migraine, chronic fatigue syndrome, fibromyalgia, depres- sion, etc.

Intriguingly Maricq and Weinrich [7] reported in 1988 that colour charts could be used to assist the diagnosis of Raynaud’s phenomenon.

In this particular case study the patient was tested using the normal SVS protocol. See Strannik Operating Manual [8] . The SVS test results indicated:

・ Systemic level: that “the regulation of blood volume” was the most destabilised physiological system (Figure 1) and that a Pathological Functional System (PFS) involving the brain and adrenal glands had been established (Figure 1).

・ Organs (Figure 1): Heart, adrenals, kidneys, blood, pituitary, thyroid and brain were identified as the most dysfunctional organs.

・ Cells (Figure 2): absence of processes associated with abnormal cell division.

・ Psycho-oncological risk of onset is associated with duodenum, ovaries, ears, nose, oesophagus, and kidneys

・ Molecular Level i.e. reported pathologies (Figures 3-13).

The most significant pathological indications reported are summarised as follows:

Note 1: the genetic signals indicate the capacity of the organ to deal with a physiological event. In general they are indicative of a genetic pre-dispo- sition and not an actual disposition i.e. it is only when the genetic condition is accompanied by a significant phenotypic indication, typically above 7 - 10 units, that this will be manifest as a pathological process and ultimately as “symptoms”.

Summary: the patient is suffering from extreme emotional stress. The consequences of this has resulted in impaired blood circulation throughout the vasculature and brain resulting in the earliest onset of portal hyperten- sion, renal insufficiency, idiopathic hypotension, nephritis, adrenal insufficiency, anaemia, encephalopathy, neu- ritis, urticaria, and is showing earliest presymptomatic onset of type 1 diabetes. These are the “acute” conditions which are characteristic of the Raynaud’s phenomenon.

In addition to the pathologies which are associated with the onset and progression of primary Raynaud’s phe- nomenon the patient currently suffers from a duodenal ulcer andthere are indications that the patient has previ- ously suffered a mild stress-related cardiovascular event/heart attack. These are the “chronic” conditions influ- encing this patient’s health.

The patient would be expected to be suffering from problems with the heart, digestion of foods, stomach pains or aches, recurrent bouts of diarrhoeia, the general inability to function, a lack of mental clarity, pins and needles in the arms and legs, cold extremities, susceptibility to infections in particular of the lungs and brochii, lack of energy and impaired ability to function.

The patient reported that they had been previously diagnosed with Raynaud’s phenomenon and that this was manifest as cold fingers and toes, “pins and needles”, and a range of recurrent medical indications. They were interested to see if SVS could determine the various pathological indications which characterised their condition. The main issues of concern to the patient was to understand the degree of dysfunction influencing the patient’s heart, lungs, peripheral nervous system, digestive system, and skin.

1) The particular medical indications reported by SVS were consistent with the patient’s known medical his- tory, their personal concerns regarding their health, and the known etiology of Raynaud’s phenomenon.

2) The fundamental problem is associated with impaired blood flow to the vasculature.

3) The pathological indications are consistent with long-term exposure to stress.

An SVS test, when taking into account the overheads of operation, cost of employing someone to administer the test, etc; is estimated at £50 - 100/test. By comparison a diagnosis of Raynaud’s disease would involve at least one consultation with the patient’s GP and several consultations with doctors and/or consultants in secondary care. It would also require long-term treatment by drugs.

It is considered to be an ideal condition for which SVS could be used to determine the onset and progression of the various Raynaud’s pathologies and for treatment by Strannik Light Therapy.

Estimated SVS Cost based upon having two GP consultations pa: £100 - 200.

It is a particular failing of modern medicine that it is poorly able to characterise and diagnose many of the com- mon lifestyle-related morbidities [16] . The emphasis upon single biochemical determinants (biomarkers) ignores the complex multi-systemic way in which the brain regulates the autonomic nervous system and physiological systems, and the even more complex range of pathological consequences which arise from autonomic dysfunc- tion. Furthermore, the emphasis upon having an individual test for each presenting co-morbidity merely adds to the complexity and cost of healthcare. There is therefore a need throughout medicine, not just regarding the diagnosis of Raynaud’s phenomenon, for a screening technology which can comprehensively and inexpensively determine and monitor the co-morbidities which present in such cases AND to better understand how the auto- nomic nervous system functions in order to treat the vast plethora of medical conditions which arise as a result of the autonomic dysfunction which arises from the effect of various stressors.

Strannik technology is the first technology to be based upon a mathematical model of the autonomic nervous system. This article highlights the potential of Strannik Virtual Scanning to simplify the task of monitoring the health of an RP patient and also to significantly reduce the cost of doing so throughout the RP patient’s life.

According to the National Health Service in the UK [17] as many as 1 in 6 of the population―an estimated 10 million of the UK population―may suffer from some degree of RP during their lives. The condition may be acute e.g. accompanying childbirth or during periods when the patient smoked cigarettes, or it may be chronic.

Accordingly this article illustrates how the cost of diagnosing the onset and progression of co-morbidities in the RP patient, and perhaps also the cost of treating RP, can be significantly reduced by using Strannik Virtual Scanning & Strannik Light Therapy [8] .

The author thanks Dr Elena Ewing MD for her assistance.

SVS: Strannik Virtual Scanning

ANS: Autonomic Nervous System

RP: Raynaud’s Phenomenon

SLT: Strannik Light Therapy