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Binder, N.B., Puchner, A., Niederreiter, B., Hayer, S., Leiss, H., Bluml, S., Kreindl, R., Smolen, J.S. and Redlich, K. (2013) Tumor necrosis factor-inhibiting therapy preferentially targets bone destruction but not synovial inflammation in a tumor necrosis factor-driven model of rheumatoid arthritis. Arthritis & Rheumatism, 65, 608-617. http://dx.doi.org/10.1002/art.37797

has been cited by the following article:

• TITLE: Multifaceted role of TNF-α during the pathogenesis of rheumatoid arthritis

  AUTHORS: Ramanjaneya V. R. Mula, Rangaiah Shashidharamurthy

  KEYWORDS: Cytokines; Tumor Necrosis Factor-α; Rheumatoid Arthritis; Inflammation

  JOURNAL NAME: Advances in Bioscience and Biotechnology, Vol.4 No.10, October 16, 2013

  ABSTRACT: Tumor necrosis factor alpha (TNF-α) a cytokine has been shown to be the key player during the pathogenesis of several autoimmune inflammatory disorders (presumably sterile inflammation) including rheumatoid arthritis (RA). Several studies have shown that TNF-α is mainly involved in the proinflammatory responses. However recent studies have reported multifunctional role of TNF-α during the development of RA. Therefore, in this article we have highlighted the distinct functions of TNF-α during pathogenesis of RA.