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Abu-Amer, Y., Ross, F.P., Edwards, J. and Teitelbaum, S.L. (1997) Lipopolysaccharide-stimulated osteoclastogenesis is mediated by tumor necrosis factor via its P55 receptor. The Journal of Clinical Investigation, 100, 1557-1565. http://dx.doi.org/10.1172/JCI119679

has been cited by the following article:

• TITLE: Multifaceted role of TNF-α during the pathogenesis of rheumatoid arthritis

  AUTHORS: Ramanjaneya V. R. Mula, Rangaiah Shashidharamurthy

  KEYWORDS: Cytokines; Tumor Necrosis Factor-α; Rheumatoid Arthritis; Inflammation

  JOURNAL NAME: Advances in Bioscience and Biotechnology, Vol.4 No.10, October 16, 2013

  ABSTRACT: Tumor necrosis factor alpha (TNF-α) a cytokine has been shown to be the key player during the pathogenesis of several autoimmune inflammatory disorders (presumably sterile inflammation) including rheumatoid arthritis (RA). Several studies have shown that TNF-α is mainly involved in the proinflammatory responses. However recent studies have reported multifunctional role of TNF-α during the development of RA. Therefore, in this article we have highlighted the distinct functions of TNF-α during pathogenesis of RA.