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H. Zhang, A. Zhang, D. E. Kohan, R. D. Nelson, F. J. Gonzalez and T. Yang, “Collecting Duct-Specific Deletion of Peroxisome Proliferator-Activated Receptor γ Blocks Thiazolidinedione-Induced Fluid Retention,” Proceedings of the National Academy of Sciences of the United States of America, Vol. 102, No. 26, 2005, pp. 9406-9411. doi:10.1073/pnas.0501744102

has been cited by the following article:

• TITLE: The Effects of Insulin Resistance and Inflammation on Renal Proximal Tubule Sodium Transport and Hypertension

  AUTHORS: Shoko Horita, Motonobu Nakamura, Masashi Suzuki, Hideomi Yamada, George Seki

  KEYWORDS: Insulin; Inflammation; TNF-α; Proximal Tubule; Insulin Resistance; PPARγ; Thiazolidinediones

  JOURNAL NAME: Open Journal of Endocrine and Metabolic Diseases, Vol.3 No.5A, August 19, 2013

  ABSTRACT: Insulin resistance, closely linked to inflammation, is recognized as a key factor in the onset and aggravation of diabetes, cardio-renal syndrome, hypertension, and obesity. In the renal proximal tubule, insulin resistance may increase renal sodium reabsorption, leading to hypertension, edema and sometimes heart failure. Recently some anti-diabetic agents have been shown to have effects on the transporters in renal proximal tubule. Because renal proximal tubule mediates about 70% of sodium reabsorption, it is quite important to clarify the function of renal proximal tubule under insulin resistance and inflammation.