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L. Viatte, G. Nicolas, D.-Q. Lou, M. Bennoun, J.-C. Lebordes-Brion, F. Cannone-Hergaux, K. Sch?nig, H. Bujard, A. Kahn, N. Andrews and S. Vaulont, “Chronic Hepcidin Induction Causes Hypoferremia and Alters the Pattern of Cellular Iron Accumulation in Hemochromatotic Mice,” Blood, Vol. 107, No. 7, 2006, pp. 2952-2958. doi:10.1182/blood-2005-10-4071

has been cited by the following article:

• TITLE: Iron-Restricted Erythropoiesis in Anaemic Patients with Giant Cell Arteritis and Polymyalgia Rheumatica

  AUTHORS: Torbjörn Karlsson

  KEYWORDS: Anaemia; Iron-Restriction; Erythropoiesis; Giant Cell Arteritis; Polymyalgia Rheumatica

  JOURNAL NAME: Open Journal of Blood Diseases, Vol.3 No.1, March 29, 2013

  ABSTRACT: The aim of this observational study was to biochemically characterize the anaemia in GCA (giant cell arteritis) and PMR (polymyalgia rheumatica) patients. Values for mean corpuscular volume, mean corpuscular hemoglobin and soluble transferrin receptor were normal, whereas serum iron and total iron binding capacity (TIBC) were subnormal, and mean ferritin was above the upper reference limit. Iron-restricted erythropoiesis (IRE), defined as a bone marrow smear staining positive for iron in combination with transferrin saturation less than 20%, was present in all patients. All patients exhibited clinical and biochemical signs of active inflammation with elevated C-reactive protein and an increased erythrocyte sedimentation rate.