Article citationsMore>>

Romeo, S., Kozlitina, J., Xing, C., Pertsemlidis, A., Cox, D., Pennachio, L.A., Boerwinkle, E., Cohen, J.C. and Hobbs, H. (2008) “Genetic variation in PNPLA3 confess susceptibility to nonalcoholic fatty liver disease. Nature Genetics, 40, 1461-1465. doi:10.1038/ng.257

has been cited by the following article:

• TITLE: Two male study groups with adiposity and hypertriglyceridemia were at risk for hypertension and alcohol use declined renal endothelium

  AUTHORS: Ruth-Maria Korth

  KEYWORDS: Adiposity; Hypertriglyceridemia; Hypertension; Alcohol Use; Dysfunctional Endothelium; Fatty Liver Disease

  JOURNAL NAME: Health, Vol.4 No.12A, December 31, 2012

  ABSTRACT: Men who attended a Bavarian General Medicine Practice were confidentially invented here. Two male study groups were enrolled to characterize adiposity or hypertriglyceridemia showing that these men were at baseline risk for hypertension [1]. Adverse alcohol consumption mediated dysfunction of renal endothelium as shown here and before [1]. This study found that alcohol use aggravated dyslipidemia, fatty liver disease and critical fasting blood glucose of obese men predicting then late hepatorenal disorders. Overall, two male study groups showed a relevant proportion of men who reported alcohol consumption showing then critical morning urines indicating dysfunction of renal endothelium. The present report looked also at healthy men who reported positive lifestyle behaviour and at men with nonalcohol adiposity and nonalcohol hypertriglyceridemia who then showed normal morning urines indicating functional renal endothelium. Relatively young men at risk were motivated to replace adverse alcohol use by healthy liquids without alcohol and by higher quality of food.