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A. S. Falcao, C. Bellarosa, A. Fernández, M. A. Brito, R. F. Silva, C. Tiribelli and D. Brites, “Role of Multidrug Ressistence-Associated Protein 1 Expression in the in Vitro Susceptibility of Rat Nerve Cell to Unconjugated Bilirubin,” Neuroscience, Vol. 144, No. 3, 2007, pp. 878-888. doi:10.1016/j.neuroscience.2006.10.026

has been cited by the following article:

• TITLE: Otoacoustic Emissions and Evoked Potentials in Infants after Breast-Feeding Jaundice——Hearing Dysfunction in Breast-Feeding Jaundice

  AUTHORS: Adrián Poblano, Norma Ballesteros, Carmina Arteaga, Blanca Flores, Teodoro Flores

  KEYWORDS: Breast-Feed Jaundice; Hyperbilirubinemia; Hearing; Neonates; Transient-Evoked Otoacoustic Emissions; Brainstem Auditory Evoked Potentials

  JOURNAL NAME: Neuroscience and Medicine, Vol.3 No.3, September 21, 2012

  ABSTRACT: We study hearing in a group of infants with Breast-feeding jaundice (BFJ) by means of Transient-evoked otoacoustic emissions (T-EOE) and Brainstem auditory evoked potentials (BAEP) searching for relationship between bilirubin serum levels and auditory dysfunction. Eleven infants born at-term with BFJ were selected for the study. We studied also 11 control age- and gender matched healthy at-term infants without signs of jaundice. T-EOAE studies were performed between 5-7 days after birth, and 3 months later. BAEP studies were performed once. BFJ group infants exhibited lower amplitudes in T-EOE than infants in the control group. These differences disappear at the 3-month evaluation. In BAEP, we observed a significant latency delay of waves I and V in Breast-feeding jaundice group infants. All infants in both groups demonstrated reproducible wave V response at 30 decibels. No significant correlation values were observed between bilirubin serum levels and T-EOE and BAEP variables. Our data suggest that BFJ can result in transient peripheral and central auditory dysfunction. Dysfunction is reversible after treatment of infants with BFJ.