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Wang, W.Y., Zhang, J., Wu, W.Y., Li, J., Ma, Y.L., Chen, W.H., Yan, H., Wang, K., Xu, W.W., Shen, J.H. and Wang, Y.P. (2011) Inhibition of lipoprotein-associated phospholipase A2 ameliorates inflammation and decreases atherosclerotic plaque formation in apoE-defocient mice. PLoS One, 6, e234-e235.

has been cited by the following article:

• TITLE: Oxidized phospholipids modify plasminogen and apolipoprotein(a): Implications for vascular disease

  AUTHORS: Angelo M. Scanu, Diana M. Stafforini, Celina Edelstein

  KEYWORDS: Plasminogen; Apolipoprotein(a); Lipoprotein(a); Oxidized Phospholipids; Oxidized Phosphatidylcholines

  JOURNAL NAME: American Journal of Molecular Biology, Vol.2 No.2, April 28, 2012

  ABSTRACT: Whereas the close structural homology between human plasminogen and apolipoprotein(a) has been known for a number of years only recent studies have revealed that both proteins carry linked oxidized phospholipids that may modify the function of these proteins. Future studies should provide a better understanding of oxidized phospholipid adducts and the role played by lipoprotein-associated phospholipase A2 for which cleavage specificity has been established when these modified lipids are in a free form.