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Fukui, M., Nakano, K., Shigeta, H., Yoshimori, K., Fujii, M., Kitagawa, Y., Mori, H., Kajiyama, S., Nakamura, N., Abe, N., Obayashi, H., Fukui, I., Ohta, K., Ohta, M. and Kondo, M. (1997) Antibodies to glutamic acid decarboxylase in Japanese diabetic patients with secondary failure of oral hypoglycaemic therapy. Diabetic Medicine, 14, 148-152. doi:10.1002/(SICI)1096-9136(199702)14:2<148::AID-DIA317>3.0.CO;2-9

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• TITLE: Reduced endogenous insulin secretion in diabetic patients with low-titer positive antibodies against GAD

  AUTHORS: Yuichiro Takeuchi, Hiroyuki Ito, Koshiro Oshikiri, Shinichi Antoku, Mariko Abe, Mizuo Mifune, Michiko Togane, Masahiro Kato

  KEYWORDS: GAD Antibody; Endogenous Insulin Secretion; Urinary C-Peptide; SPIDDM; LADA

  JOURNAL NAME: Journal of Diabetes Mellitus, Vol.2 No.1, February 22, 2012

  ABSTRACT: Aim: To investigate the clinical characteristics of diabetic patients with a low-titer positive for the anti-glutamic acid decarboxylase 65 antibody (GAD antibody). Methods: The subjects were 420 diabetic inpatients. The endogenous insulin secretion was estimated on the basis of the C-peptide immunoreactivity from a 24 h urine collection (uCPR). Clinical variables were compared between patients negative for the GAD antibody (GAD antibody titer