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Hagopian, W.A., Karlsen, A.E., Gottsater, A., Landin-Olsson, M., Grubin, C.E., Sundkvist, G., Petersen, J.S., Boel, E., Dyrberg, T. and Lernmark, A. (1993) Quantitative assay using recombinant human islet glutamic acid decarboxylase (GAD65) shows that 64K autoantibody positivity at onset predicts diabetes type. The Journal of Clinical Investigation, 91, 368-374. doi:10.1172/JCI116195

has been cited by the following article:

• TITLE: Reduced endogenous insulin secretion in diabetic patients with low-titer positive antibodies against GAD

  AUTHORS: Yuichiro Takeuchi, Hiroyuki Ito, Koshiro Oshikiri, Shinichi Antoku, Mariko Abe, Mizuo Mifune, Michiko Togane, Masahiro Kato

  KEYWORDS: GAD Antibody; Endogenous Insulin Secretion; Urinary C-Peptide; SPIDDM; LADA

  JOURNAL NAME: Journal of Diabetes Mellitus, Vol.2 No.1, February 22, 2012

  ABSTRACT: Aim: To investigate the clinical characteristics of diabetic patients with a low-titer positive for the anti-glutamic acid decarboxylase 65 antibody (GAD antibody). Methods: The subjects were 420 diabetic inpatients. The endogenous insulin secretion was estimated on the basis of the C-peptide immunoreactivity from a 24 h urine collection (uCPR). Clinical variables were compared between patients negative for the GAD antibody (GAD antibody titer