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Montaner, S., Sodhi, A., Molinolo, A., Bugge, T.H., Sawai, E.T., et al. (2003) Endothelial Infection with KSHV Genes in Vivo Reveals that vGPCR Initiates Kaposi’s Sarcomagenesis and Can Promote the Tumorigenic Potential of Viral Latent Genes. Cancer Cell, 3, 23-26.
https://doi.org/10.1016/S1535-6108(02)00237-4

has been cited by the following article:

• TITLE: Kaposi Sarcoma after Kidney Transplant: A Clinical Case Report

  AUTHORS: Meryem Benbella, Zaineb Kaouiri, Imane Saidi, Naima Ouzedoun, Tarik Bouattar

  KEYWORDS: Kaposi, Kidney Transplant, HHV8, Immunosuppression

  JOURNAL NAME: Open Journal of Nephrology, Vol.14 No.1, March 29, 2024

  ABSTRACT: Kaposi sarcoma is a neoplasm caused by human herpesvirus 8 (HHV8) that most commonly affects immunosuppressed patients. The skin is the most affected area, but other sites can be involved such as the lung, digestive tract and lymph nodes. The classical presentation involves a violaceous skin lesion that can be small or hidden, leading to a delay in diagnosis. We report a clinical case of a kidney transplant patient, who presented, 14 months after kidney transplant, with unilateral edema of the inferior member and cutaneous rash misdiagnosed and taken initially for erysipelas. The diagnosis of Kaposi’s sarcoma was retained, on a lymph node biopsy of an inguinal adenopathy. The evolution was marked by a local and general improvement after systemic chemotherapy, reducing Tacrolimus and discontinuation of Mycophenolate mofetil. Graft function remained stable during the follow-up.