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Yoshida, M., Shiojima, I., Ikeda, H., et al. (2009) Chronic Doxorubicin Cardiotoxicity is Mediated by Oxidative DNA Damage-ATM-p53-Apoptosis Pathway and Attenuated by Pitavastatin through the Inhibition of Rac1 Activity. Journal of Molecular and Cellular Cardiology, 47, 698-705.
https://doi.org/10.1016/j.yjmcc.2009.07.024

has been cited by the following article:

• TITLE: Effect of Ganoderma lucidum Polysaccharide on Antioxidative Ability of Rats with Myocardial Injury Induced by Doxorubicin

  AUTHORS: Fan Xu, Xiao Li, Qingshan Li, Xiao Xu, Wenxin Li, Xiaolei Yu

  KEYWORDS: Doxorubicin, Ganoderma lucidum Polysaccharide, Myocardial Tissue, Antioxidant Capacity

  JOURNAL NAME: Journal of Biosciences and Medicines, Vol.10 No.4, March 31, 2022

  ABSTRACT: Objective: The objective is to investigate the effect of Ganoderma lucidum polysaccharide (GLPS) on the oxidative stress induced by doxorubicin (DOX) in cardiomyocytes. Methods: SD rats were divided into control group, DOX group, GLPS low dose and high dose + DOX group. SOD and MDA levels in myocardial tissue were detected in each group. The expression of related proteins in each group was detected by Western blot. Results: GLPS can increase the SOD level and decrease MDA caused by DOX (p Conclusion: Ganoderma polysaccharide can improve the oxidative stress injury of myocardial tissue induced by doxorubicin and play a protective role in myocardial tissue.