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Verrier, R.L., Bonatti, R., Silva, A.F., Batatinha, J.A., Nearing, B.D., Liu, G., et al. (2014) If Inhibition in the Atrioventricular Node by Ivabradine Causes Rate-Dependent Slowing of Conduction and Reduces Ventricular Rate during Atrial Fibrillation. Heart Rhythm, 11, 2288-2296.

has been cited by the following article:

• TITLE: Ivabradine—The Final Crusader for Postoperative Junctional Ectopic Tachycardia, a Case Report with Literature Review

  AUTHORS: Manoj Kumar Sahu, Harsha Vardhan Niraghatam, Nikhil Bansal, Sarvesh Pal Singh, Palleti Rajashekar, Shiv Kumar Choudhary

  KEYWORDS: Tetralogy of Fallot, Junctional Ectopic Tachycardia, Ivabradine

  JOURNAL NAME: World Journal of Cardiovascular Surgery, Vol.9 No.8, August 22, 2019

  ABSTRACT: Background: Junctional ectopic tachycardia (JET) is one troublesome supraventricular arrhythmia in postoperative pediatric cardiac surgical patients. Unless treated timely and effectively it may lead to morbidity and even mortality. Aim: To understand the role of Ivabradine in the treatment of JET in postoperative pediatric cardiac surgical patients. Case: We present a postoperative case of complete repair of Tetralogy of Fallot who was in normal sinus rhythm in the preoperative period and developed JET 4 hours later in postoperative period which was persistent despite measures to optimize the internal milieu of the body and conventional antiarrhythmics, that was successfully treated with Ivabradine at a dose of 0.05 mg/kg/12 hourly. Conclusion: Ivabradine can be used to successfully treat postoperative JET in cases ofrefractory to other antiarrhythmic drugs like Digoxin and Amiodarone.