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Wijese, K.N., Dai, F.F., Hardy, A.B., Giglou, P.R., Bhattacharjee, A., Koshkin, V., Chimienti, F., Gaisano, H.Y., Rutter, G.A. and Wheeler, B.B. (2010) Beta Cell Specific ZnT8 Deletion in Mice Causes Marked Defects in Insulin Processing Crystallization and Secretion. Diabetologia, 53, 1656-1668.
https://doi.org/10.1007/s00125-010-1733-9

has been cited by the following article:

• TITLE: Biochemical Assessment of the Relationship of Zinc and Glucose Concentration in Diabetics

  AUTHORS: Gborienemi Simeon George, Osagie Endely Friendgood

  KEYWORDS: Biochemical, Relationship, Zinc, Glucose, Diabetics

  JOURNAL NAME: Open Journal of Endocrine and Metabolic Diseases, Vol.8 No.4, April 30, 2018

  ABSTRACT: Zinc plays a critical role in a variety of cell functions and elicits the fact that both its deficiency and excess may demonstrate deleterious situation. It has been proposed that zinc is required for multiple steps in insulin synthesis and release. This study investigated the relationship of zinc and glucose in diabetics. Using serum samples of diabetic patients whose glucose concentrations were above the threshold (10.0 mmol/l), spectroscopic methods were used to determine the concentration of glucose and zinc. Results obtained showed a negative correlation between level of glucose and zinc. Data were analyzed using student’s t-test with the aid of Graph Pad Prism (R) version 6.01 with a p value of