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Clark, J.A., Turner, M.L., Howard, L., Stanescu, H., Kleta, R. and Kopp, J.B. (2009) Description of Familial Keloids in Five Pedigrees: Evidence for Autosomal Dominant Inheritance and Phenotypic Heterogeneity. BMC Dermatology, 9, 8.
http://dx.doi.org/10.1186/1471-5945-9-8
Xai, W., Phan, T.T., Lim, I.J., Longaker, M.T. and Yang, G.P. (2006) Differential Transcriptional Responses of Keloid and Normal Keratinocytes to Serum Stimulation. Journal of Surgical Research, 135, 156-163.
http://dx.doi.org/10.1016/j.jss.2006.01.031

has been cited by the following article:

• TITLE: Familial Keloid in Indian Scenario: Case Report and Review of Literature

  AUTHORS: Snigdha Goyal, Isha Saini, Sunder Goyal

  KEYWORDS: Familial Keloid, Autosomal Dominant, Autosomal Recessive

  JOURNAL NAME: Open Access Library Journal, Vol.2 No.7, July 21, 2015

  ABSTRACT: Keloid disease is a fibro proliferative skin tumor and occurs after a skin trauma in genetically vulnerable individuals. Etiology of keloid is unclear. Causative factors for keloid are increased familial aggregation, a higher prevalence in certain races, parallelism in identical twins, and alteration in gene expression. It appears that the surroundings activate the disease in genetically susceptible individuals. Several genes are considered responsible for keloid disease, but no single gene mutation has thus far been found to be responsible. Therefore, we should apply a combination of methods such as association, gene-gene interaction, epigenetics, linkage, gene expression, and protein analysis to find out the keloid etiology. Incidence of familial keloid is common in Africans but is uncommon in Indian population. It may be due to autosomal dominant or autosomal recessive inheritance.