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Pizzimenti, S., Ciamporcero, E., Daga, M., Pettazzoni, P., Arcaro, A. Cetrangolo, G., Minelli, R., Dianzani, C., Lepore, A., Gentile, F. and Barrera, G. (2013) Interaction of Aldehydes Derived from Lipid Peroxidation and Membrane Proteins. Frontiers in Physiology, 4, 242.

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• TITLE: LDL-Related Intolerance to Glucose, Diastolic Hypertension and Additive Effects of Smoking Were Found with Three Female Study Groups

  AUTHORS: Ruth-Maria Korth

  KEYWORDS: Combined Telemedical Care: Women’s Health, Obesity, LDL-Intolerance to Glucose, Diastolic Hypertension, Ratio of Serum Albumin to Triglycerides (Alb/Trig), Albuminuria

  JOURNAL NAME: Health, Vol.8 No.3, February 19, 2016

  ABSTRACT: Initial prodiabetic risk profiles were invented here with three female study groups consisting of primarily healthy women (A1: 1990-1999, n = 160; A2: 2009, n = 88; A: n = 248, 36 ± 14 years; B: 2014: n = 65, aged 37± 11 years). Significantly higher blood pressure was found comparing intolerance versus tolerance to glucose (p p p = 0.02), of fasting blood glucose (p = 0.07) and of urine pathology (p = 0.07). High LDL-C of women who reported smoking at baseline was correlated with diastolic hypertension whereby alcohol problems overlapped (p = 0.036, A). Unhealthy combinations were found consisting of LDL-related intolerance to glucose, LDL-related smoking, of alcohol-related hypertriglyceridemia or of combined drinking and smoking testing urine pathology over the course of time. Obese women were at direct risk for hypertension in the presence of high LDL-C and submaximal ratio of serum albumin to triglycerides (Alb/Trig). Obese women reacted highly sensitive to critical alcohol consumption showing then macroalbuminuria. Current participants who disowned daily alcohol consumption showed healthy morning urines and normal fasting blood glucose. Mild decrease of HDL-C was observed during heavy smoking of relatively young women who had normal biomarkers. Women with intolerance to glucose were at direct risk for hypertension whereby high LDL-C and/or smoking triggered prodiabetic risk profiles. Obese women had elevated LDL-C during hypertension and reacted highly sensitive to alcohol-related proteinuria and/or hematuria.