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Royer, S. and Pare, D. (2002) Bidirectional Synaptic Plasticity in Intercalated Amygdale Neurons and the Extinction of Conditioned Fear Responses. Neuroscience, 115, 455-462.
http://dx.doi.org/10.1016/S0306-4522(02)00455-4

has been cited by the following article:

• TITLE: Differential Effect of Unilateral Amygdalar GABAA Receptor Agonist Injection on Low- and High-Anxiety Rats

  AUTHORS: Maria P. Rysakova, Irina V. Pavlova

  KEYWORDS: Anxiety, Basolateral Amygdala, GABAA Receptor, Muscimol Hydrobromide, Hemispheric Lateralization

  JOURNAL NAME: Journal of Behavioral and Brain Science, Vol.6 No.1, January 15, 2016

  ABSTRACT: The influence of γ-aminobutyric type-A (GABAA) receptors agonist (muscimol hydrobromide, 0.1 μg/0.5 μl) injections into the right or left basolateral amygdala (BLA) on the behavior of high-anxiety (HA) and low-anxiety (LA) rats subjected to the elevated plus-maze (EPM) test was investigated. Anxiolytic-like effects (increase of open-arm entries and open-arm time) was revealed only after administration of muscimol into the left (but not right) amygdala of HA animals. No effect was observed upon administration of muscimol to LA rats. These findings suggest an important role in anxiety regulation of the amygdalar GABA levels, and the assumed GABA hemispheric lateralization.