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Brown, D.A., Chicco, A.J., Jew, K.N., Johnson, M.S., Lynch, J.M., Watson, P.A. and Moore, R.L. (2005) Cardioprotection Afforded by Chronic Exercise Is Mediated by the Sarcolemmal, and Not the Mitochondrial, Isoform of the KATP Channel in the Rat. The Journal of Physiology, 569, 913-924.
http://dx.doi.org/10.1113/jphysiol.2005.095729

has been cited by the following article:

• TITLE: Effects of mRNA, Protein Expression and Activity for Myocardial SOD2 by a Single Bout of or Long-Term Strenuous Endurance Exercise in Rats

  AUTHORS: Simao Xu, Weichun Liu, Minhua Li

  KEYWORDS: SOD2, Strenuous Endurance Exercise, Myocardia

  JOURNAL NAME: Chinese Medicine, Vol.5 No.4, December 19, 2014

  ABSTRACT: Objective: To explore the effects of myocardial SOD2 by strenuous endurance exercise. Methods: 27 grown male SD rats were randomly divided into control group (C), a single bout of strenuous endurance exercise group (E1) and seventh-week strenuous endurance exercise group (E2). Real-time PCR was used to observe the changes of mRNA expression for myocardial SOD2. Western bolt was used to observe the changes of SOD2 protein expression. In addition, SOD2, T-SOD and SOD1 activity changes were observed. Results: Myocardial SOD2 expression level at mRNA and protein of Group E1, E2 was significantly higher than that in group C, and SOD2 and T-SOD activity in group E2 were significantly higher than those in group C. Those changes were more obvious in group E2. Conclusions: Strenuous endurance exercise can improve level of myocardial SOD2 expression at mRNA and protein, and enhance the activity for SOD2, thus increasing the activity for T-SOD. Effect of long-term strenuous endurance exercise was better than a single bout of one.