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Ait El Kadi, M., Aghrouch, M., Seffar, M., Elharti, J., Bouklouze, A., Cherrah, Y., et al. (2006) Prévalence des souches d'Acinetobacter baumannii et de Pseudomonas aeruginosa résistantes à l'imipénème par production de métallo-β-lactamases. Med Mal Infect., 36, 386-389.
http://dx.doi.org/10.1016/j.medmal.2006.05.003

has been cited by the following article:

• TITLE: Resistance to Beta Lactam Antibiotics of Pseudomonas aeruginosa Isolated in Community Infections within HIV Infected Persons in Lomé-Togo

  AUTHORS: Majesté Ihou Wateba, Kanyni Ekoue-Kouvahey, Abago Balaka, Komivi Tsatsu, Ousseni Tidjani

  KEYWORDS: HIV-1; Pseudomonas aeruginosa; Resistance Phenotypes; Beta Lactam Antibiotic

  JOURNAL NAME: World Journal of AIDS, Vol.4 No.1, March 12, 2014

  ABSTRACT: Objective: Describe resistance to beta lactam antibiotics of Pseudomonas aeruginosa in community infection within HIV-1 infected persons. Methods: We have studied prospectively from June 15th to December 31st 2013 inthe Clinic Hotel-Dieu and NGO VISA of Lomé, adult HIV-1 infected patients under anti retroviral therapy combining tenofovir, lamivudine and efavirenz for at least one year. The technique of agar diffusion susceptibility using discs of Ticarcillin + clavulanic acid is used to study the β-lactamase production. The diagnosis of species was performed by the chloroform test and the test for sensitivity to kanamycin and colistin. Results: Thirty five strains of Pseudomonas aeruginosa were obtained. The T-lymphocytes CD4 mediane was 575 cells/mm3 of blood. Urine represented 15 cases, skin abscesses 11 cases, externa suppurate otitis 7 cases and vaginal swab for 2 cases. The phenotypes were: wild phenotypes 23 cases (65.7%), resistant phenotypes 12 cases. Among resistant phenotypes, 4 were complex phenotype; 5 were ESBL phenotypes; 2 were hyper productive cephalosporinases phenotypes and 1 was a specific phenotype with impermeability to imipenem. Conclusion: The acquisition of resistance of Pseudomonas aeruginosa to beta lactam antibiotics in community infections among HIV-1 infected person incentives controls and promotes the rational use of antibiotics.