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Roberson, E.D., Halabisky, B., Yoo, J.W., Yao, J., Chin, J., Yan, F., Wu, T., Hamto, P., Devidze, N., Yu, G.Q., Palop, J.J., Noebels, J.L. and Mucke, L. (2011) Amy-loid-Beta/Fyn-Induced Synaptic, Network, and Cognitive Impairments Depend on Tau Levels in Multiple Mouse Models of Alzheimer’s Disease. The Journal of Neuroscience, 31, 700 -711. http://dx.doi.org/10.1523/JNEUROSCI.4152-10.2011

has been cited by the following article:

• TITLE: Mapping It out: A Novel Signaling Pathway Linking Aβ-PrPc-Fyn Complex to Cognitive Impairment in Alzheimer’s Disease

  AUTHORS: Yiling Wang, Lei Xue

  KEYWORDS: Alzheimer’s; PrPc; Aβ; Fyn; Tau; Microtubule

  JOURNAL NAME: Advances in Alzheimer's Disease, Vol.3 No.1, March 12, 2014

  ABSTRACT: Understanding the signaling cascade that leads to the rapid memory and cognitive breakdown in Alzheimer’s disease is the key to finding a potential treatment method for the disease. Recently, Larson et al. connect the roles of major proteins implicated in the disease progression and propose targeting of cellular prion protein (PrPc) as a way of someday rescuing synaptic plasticity in humans with Alzheimer’s, as it has been done on mice.