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Shankar, G.M., Li, S., Mehta, T.H., Garcia-Munoz, A., Shepardson, N.E., Smith, I., Brett, F.M., Farrell, M.A., Rowan, M.J., Lemere, C.A., Regan, C.M., Walsh, D.M., Sabatini, B.L., and Selkoe, D.J. (2008) Amyloid β-Protein Dimers Isolated Directly from Alzheimer Brains Impair Synaptic Plasticity and Memory. Nature Medicine, 8, 837-842. http://dx.doi.org/10.1038/nm1782

has been cited by the following article:

• TITLE: Mapping It out: A Novel Signaling Pathway Linking Aβ-PrPc-Fyn Complex to Cognitive Impairment in Alzheimer’s Disease

  AUTHORS: Yiling Wang, Lei Xue

  KEYWORDS: Alzheimer’s; PrPc; Aβ; Fyn; Tau; Microtubule

  JOURNAL NAME: Advances in Alzheimer's Disease, Vol.3 No.1, March 12, 2014

  ABSTRACT: Understanding the signaling cascade that leads to the rapid memory and cognitive breakdown in Alzheimer’s disease is the key to finding a potential treatment method for the disease. Recently, Larson et al. connect the roles of major proteins implicated in the disease progression and propose targeting of cellular prion protein (PrPc) as a way of someday rescuing synaptic plasticity in humans with Alzheimer’s, as it has been done on mice.